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Trial ‘soon’ for aid worker who facilitated release of Italian journalist in Afghanistan
Mar 17

Wednesday, May 9, 2007

An Afghan doctor, Rahmatullah Hanefi, who works for the Italian non-governmental organization (NGO), Emergency, will go on trial in Afghanistan for his alleged role in the March, 2007 slaying of Afghan interpreter and reporter, Adjmal Nashbandi.

Italian journalist, Daniele Mastrogiacomo, his driver and interpreter were kidnapped by the Taliban in Helmond Province, Afghanistan on March 5, 2007. During the ensuing negotiations for Mastrogiacomo’s release, his driver, Sayed Agha, was killed by their Taliban captors. Mastrogiacomo’s interpreter, Adjmal Nashbandi, was later executed by the Taliban. The release of Mastrogiacomo was controversial and criticized at the time for the exchange of five Taliban prisoners in return. Hanefi mediated negotiations, reportedly, with the Taliban for the release of Mastrogiacomo.

Afghanistan’s ambassador in Rome said Tuesday that Hanefi would be put on trial “soon”. It was not revealed what the specific charges against Hanefi are, but it has been suggested that the offence may be accessory to murder in the death of Nashbandi. “Hanefi’s case will come to trial soon, maybe in a week, and a lawyer will be nominated to assist him,” said Musa Maroufi, Afghanistan’s ambassador to Italy. He added that “no execution will be carried out” against Hanefi.

In April, Emergency closed the three hospitals it operates in Afghanistan. The NGO has denied that Hanefi had anything to do with Nashbandi’s death. In a news update on its U.S. website Monday, Emergency suggested that their international staff were forcibly removed from the hospitals by Afghan authorities.

“Continuing under a pretense of normalcy in this situation would provide services so inadequate as to endanger our patients,” said Emergency in the news update. They have decided “to suspend all activity until conditions allowed for the return of… international staff” and suggested a clarification was required from the Afghan government “concerning intimidating threats” allegedly made by the government.

Emergency is pressing for the release of Hanefi.

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Wayne Rooney will play in Germany
Mar 16

Wednesday, June 7, 2006

England soccer player Wayne Rooney, 20, has been passed fit to play in the FIFA World Cup soccer tournament which starts on June 9 in Germany. The £27 million Manchester United star had a scan on Wednesday that showed the 4th metatarsal he broke on Apr 29 had healed – in less than 6 weeks.

Rooney was supposedly spotted boarding a plane to Germany while the BBC claim sources close to the Football Association indicated Rooney was in England’s final World Cup squad.

The news is considered a big boost to England’s chances to win the World Cup. Manchester United manager Sir Alex Ferguson had said the player’s chances of playing were “a forlorn hope.”

The English soccer club, recently bought by American billionaire Malcolm Glazer, was concerned the investment would risk a worse injury if the previous toe-bone break had not healed when he started to play again. Manchester United’s club doctor Todd Gill and physiotherapist Rob Swire, accompanied by a small army of Red Devils’ lawyers, were said to have attended the foot scan.

The scan was considered so important to England’s fortunes that Eriksson sent England’s doctor to observe the event. A FIFA official was also present in the crowd at the moment of truth for Rooney’s foot. The official would have been final decision-maker in case of a dispute between one of the richest soccer clubs in the world and the country that invented the game.

The Swedish manager of the English football team Sven-Göran Eriksson, who had said Rooney would play some part in the competition, named Rooney in England’s 23-man squad for Germany.

The World Cup tournament pits soccer players from national teams against each other for a trophy. Previously graced by Pele and Maradona, England has not won the competition for 40 years.

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New Zealand dog saves five children; receives international attention
Mar 16

Saturday, May 5, 2007

A small, “nicely trained” Jack Russell Terrier gave his life to help save a group of five children from two aggressive pitbulls last Sunday in Manaia, Taranaki, New Zealand.

The dog named George is being described as a hero, gathering a huge amount of international media interest in the process.

The dog suffered massive injuries that the vet had never seen to that extent before, which resulted in 69-year-old Alan Gay, the dog’s owner, allowing George to be put down. He now regrets this decision.

The two pitbulls rushed at the group of children that George was following to the dairy, including a four-year-old. George then started barking loudly at the pair of dogs, and put himself between the dogs and the children. Mr Gay told Fox News, “If it wasn’t for George, those kids would have copped it.”

Despite having received offers of new dogs, Mr Gay has said that he will wait a while before getting another Jack Russell Terrier, as he is afraid it could happen again.

It is also believed that the two attacking dogs, which have been destroyed, were bred to be aggressive, including being fed the drug methamphetamine, commonly referred to as “P” in New Zealand.

Mr Gay has said that Manaia has had a problem with stray dogs before, but he never expected that George would become a victim.

One story ran in USA Today, which prompted over 120 comments, and one reader to personally call Alan Gay expressing their condolences.

Responding to the huge amount of media interest, Mr Gay said, “This really surprises me, and it’s marvellous. I never expected this … I’m surprised it got around the world as it did.” He says he has been getting a huge amount of phone calls from the media and the public. “The phone has been going since about half past seven this morning. Every time I hang up it rings again. It’s worn out; I might have to get a new one.”

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News briefs:April 16, 2010
Mar 14
Wikinews Audio Briefs Credits
Produced By
Turtlestack
Recorded By
Turtlestack
Written By
Turtlestack
Listen To This Brief

Problems? See our media guide.

[edit]

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Home of Stonehenge builders found
Mar 14

Tuesday, January 30, 2007

Scientists have uncovered the largest Neolithic settlement in the United Kingdom at the Durrington Walls and believe that the village was inhabited by the people who built the Stonehenge monument.

Scientists say that the village was built around 2,600 B.C., roughly when Stonehenge was believed to have been constructed, and housed over 100 people.

Inside the areas which would have been the interior of houses at the time, scientists also found outlines of what they think were beds and cupboards or dressers. Pieces of pottery and “filthy” rubbish around the site. Animal bones, arrowheads, stone tools and other relics were also discovered.

“We’ve never seen such quantities of pottery and animal bone and flint. In what were houses, we have excavated the outlines on the floors of box beds and wooden dressers or cupboards,” said Sheffield University archaeologist Mike Parker Pearson.

So far, the dig has revealed at least 8 houses roughly 14-16 feet square, but scientists say that they think there may have been at least 25 altogether.

The site was likely to have been occupied only seasonally rather than year-round and evidence suggests that a lot of “partying” went on at the location.

“The animal bones are being thrown away half-eaten. It’s what we call a feasting assemblage. This is where they went to party – you could say it was the first free festival. The rubbish isn’t your average domestic debris. There’s a lack of craft-working equipment for cleaning animal hides and no evidence for crop-processing,” added Pearson.

The Durrington Walls are approximately 2 miles from the Stonehenge site.

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News briefs:June 4, 2010
Mar 14
Wikinews Audio Briefs Credits
Produced By
Turtlestack
Recorded By
Turtlestack
Written By
Turtlestack
Listen To This Brief

Problems? See our media guide.

[edit]

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BellSouth denies phone records were handed over to the NSA
Mar 11

Tuesday, May 16, 2006

BellSouth, said in a statement yesterday that the telecommunications company did not hand over customer call records to the National Security Agency or NSA. On May 11, 2006, USA Today reported that the NSA collected millions of call logs from telecommunications companies in 2001 under a contract the NSA claims to have had with the company.

BellSouth said that they conducted a “internal review” and that the review “confirmed no such contract exists and we have not provided bulk customer calling records to the NSA,” said Jeff Battcher, spokesman for BellSouth.

“We do not believe that any final review will turn up anything different from what we have currently found. There is no link between the NSA and BellSouth that we can find in what we feel is a very exhaustive review. We wouldn’t have made this bold statement if we weren’t confident about this,” added Battcher.

AT&T and Verizon Communications were also said to have handed over customer call logs, but Verizon said on Friday that they don’t “and will not, provide any government agency unfettered access to our customer records or provide information to the government under circumstances that would allow a fishing expedition.”

On Monday, Massachusetts Representative Edward Markey of the House Energy and Commerce subcommittee on telecommunications asked for help from the Federal Communications Commission or FCC, to investigate whether Verizon, AT&T, and BellSouth violated privacy rights under communication laws and regulations.

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British surfers catch more than waves: Scientists find antibiotic-resistant bacteria
Mar 10

Wednesday, January 17, 2018

In findings published Sunday in Environmental International, a team from Britain’s University of Exeter reports that surfers and bodyboarders are roughly three times as likely to house antibiotic-resistant Escherichia coli and other bacteria that could easily become resistant, than other people who recreate in the coastal waters of the United Kingdom.

The epidemiological study was nicknamed the “Beach Bum Survey”. The project was performed on 143 regular surfers, body surfers and bodyboarders from around the United Kingdom. Each surfgoing participant was asked to recruit a non-surfing friend of the same sex and approximate age and who lived in the same part of the country to serve as a control, which resulted in a control group of 130.

All participants mailed in rectal swabs, and the researchers cultured the E. coli from those samples with a common antibiotic called cefotaxime. The antibiotic failed to kill the bacteria in about 9% (13 out of 143) of surfer and bodyboarder samples and 3% of the samples from the control group (4 out of 130). A deeper look into the genomes of the specific strains of bacteria found in the study volunteers showed that bacteria from surfers were four times as likely to possess genes that can be transferred from one bacterial strain to another, which can help antibiotic-susceptible bacteria become resistant. The study also involved examination of water samples from the coasts of England and Wales to estimate the risk of surfers and other beachgoers ingesting E. coli.

E. coli is a regular resident in the guts of humans and other animals. Most strains are harmless but others can cause serious disease. Like other bacteria, E. coli can undergo horizontal gene transfer, swapping genes from one bacterium to another. This can give the altered strain the ability to cause disease, survive in the presence of antibiotics or both.

Although the researchers expressed concern surfers might spread dangerous bacteria, Dr. Will Gaze, the University of Exeter Medical School professional who supervised the project, urged people not to avoid the beach: “We are not seeking to discourage people from spending time in the sea, an activity which has a lot of benefits in terms of exercise, well-being and connecting with nature”, he said. “It is important that people understand the risks involved so that they can make informed decisions about their bathing and sporting habits. We now hope that our results will help policy-makers, beach managers, and water companies to make evidence-based decisions to improve water quality even further for the benefit of public health.”

David Smith, science and policy officer of Surfers Against Sewage, which helped organise the volunteers, agreed the study was not meant to discourage surfing: “Water quality in the UK has improved vastly in the past 30 years and is some of the cleanest in Europe. Recognising coastal waters as a pathway for antibiotic resistance can allow policy makers to make changes to protect water users and the wider public from the threat of antibiotic resistance.”

One of the principal findings of this work was that existing methods may have been underestimating the prevalence of these bacteria in seawater. Previous studies have shown that even designated swimming beaches can be affected by runoff from farms or even sewage, and surfers swallow roughly ten times as much seawater as swimmers. Professor Colin Gardner of the charity Antibiotic Research UK says, these forms of runoff can have even higher concentrations of antibiotics than patients undergoing antibiotic treatment. “Research into new medicines to replace our archaic antibiotics has stagnated and unless new treatments are found, this could be potentially devastating for human health”, he warns.

The World Health Organization has reported that because so many kinds of bacteria are gaining resistance to common medicines, conditions such as pneumonia and gonnorhea may become more difficult to treat and have higher rates of sickness and death. Doctors often prescribe preventative antibiotics to patients undergoing surgery or radiation therapy, and this may also be impacted. Professor Sally Davies, England’s chief medical officer, has described a “post-antibiotic apocalypse” scenario

The European Regional Development Fund and Natural Environment Research Council provided funding for the study.

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Wikinews interviews DuckDuckGo, Opera, Mozilla, Wikimedia about DoNotTrack feature
Mar 10
This article mentions the Wikimedia Foundation, one of its projects, or people related to it. Wikinews is a project of the Wikimedia Foundation.

Tuesday, February 5, 2013

Following the introduction of a “Do Not Track” feature in modern browsers at the end of last year, Wikinews interviewed several companies and groups about the feature.

Contents

  • 1 DuckDuckGo
  • 2 Opera
  • 3 Mozilla
  • 4 Wikimedia
  • 5 Sources
  • 6 External links
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Australian researchers confirm stress makes you sick
Mar 10

Wednesday, December 7, 2005

Australian researchers say they have scientifically proven that stress causes sickness. The Garvan Institute in Sydney has discovered that a hormone, known as neuropeptide Y, (NPY) is released into the body during times of stress. Their findings show the hormone can stop the immune system from functioning properly.

Neuropeptide Y is one of those hormones that gets unregulated or released from neurones when stressful situations occur…it’s known for example that it regulates blood pressure and heart rates so your heart rate goes up but it hasn’t been known that it actually can affect immune cells as well,” said Professor Herbert Herzog, one of the researchers.

Herzog feels it is good to finally have proof of something people have suspected for so long.

“Now we have proven without doubt that there is a direct link and that stress can weaken the immune system and that makes you more vulnerable when you for example have a cold or flu and even in the more serious situations such as cancer can be enhanced in these situations,” said Herzog.

The Garvan Institute study centres on two key events that enable the human body to recognise foreign substances and control invaders. When our body encounters a pathogen (bacteria and viruses), the immune cells retain and interrogate suspects. Their activation is made possible by NPY. These cells then return to the lymph nodes, which are found all over the body, with information about the foreign invaders. The lymph nodes are where decisions about defence are made.

“Most of us expect to come down with a cold or other illness when we are under pressure, but until now we have mostly had circumstantial evidence for a link between the brain and the immune system,” said lead Garvan researcher, associate Professor Fabienne Mackay. “During periods of stress, nerves release a lot of NPY and it gets into the bloodstream, where it directly impacts on the cells in the immune system that look out for and destroy pathogens (bacteria and viruses) in the body.”

In the case of bacteria and viruses, TH1 cells are part of the attack team that is sent out on the ‘search and destroy’ mission. But when their job is done they need to be turned ‘off’ and the immune system reset. The same hormone, NPY, that activates the sentry cells now prompts the TH1 cells to slow down and die.

“Under normal conditions, circulating immune cells produce small amounts of NPY, which enables the immune cells on sentry duty and the TH1 immune cells to operate – it’s a yin and yang kind of situation. But too much NPY means that the TH1 attack is prevented despite the foreign invaders being identified – and this is what happens during stress,” added McKay.

The impact of stress on the body has been observed in athletes. Ph. D researcher at the University of Queensland, Luke Spence, together with the Australian Institute of Sport, studied elite and recreational athletes over five months.

They found elite athletes were more susceptible to respiratory diseases under stress.

“A lot of elite athletes put themselves through vast amounts of physical stress in their training, but also their emotional, psychological stress of feeling the pressure of Australia on their shoulders, wanting to compete and wanting to do their best,” said Spence.

It’s not just athletes who are prone to stress. Pressures at work and at home may cause emotional and mental stress that can be equally damaging. Almost a third of all work absenteeism in Australia is due to illness, costing employers over $10 billion a year.

“I think it has a huge impact for the work force and also for employers – if their employees are constantly stressed, constantly under pressure, they are more likely to get sick,” Spence said.

Further research could lead to the development of new drugs which may inhibit the action of the neuropeptide Y hormone.

Herzog warns people to minimise stress before it becomes a problem.

“Relaxation methods like yoga will help you to prevent that but there will still be people out there that are not responding to that and treatment by interfering with the system will be important,” he said. “There’s obviously some time until such a treatment will be available but this is something we will definitely work towards.”

The Garvan research will be published in the Journal of Experimental Medicine, Volume 202, No. 11.

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